Talk:Paroxysmal supraventricular tachycardia
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Catheter ablation
editFor recurrent of persistent SVT, we should mention catheter ablation, as for atrial fibrillation. Dan Woodard MD Danwoodard (talk) 11:10, 18 September 2018 (UTC)
Specifically upon waking?
edit(Yeah, I know - WP isn't for medical advice..) Is there any reference out there about tachycardia specifically (but sporadically) an immediately upon waking, even before rising (so not some kind of orthostatic tachycardia), with no other symptoms? Jimw338 (talk) 16:54, 5 July 2017 (UTC)
- There are lots of other types of tachycardia Doc James (talk · contribs · email) 05:19, 6 July 2017 (UTC)
Review the Anatomy section
editCould you please review the anatomy section of the article on paroxysmal supraventricular tachycardia, particularly the description of the ECG image? I added this section as a whole, although I'm not a cardiologist. Thank you! I also edited the paroxysmal supraventricular tachycardia in many other places. If you could, please then review the whole article. Thank you in advance! Maxim Masiutin (talk) 12:45, 7 November 2023 (UTC)
- Iztwoz, can you please help review the section and the image caption in this section? Maxim Masiutin (talk) 18:41, 14 November 2023 (UTC)
- Have made a few edits but I am no expert. Best Iztwoz (talk) 11:28, 15 November 2023 (UTC)
- Why did you remove the following explanation "Lead II of electrocardiogram shows 3 rapid heartbeats, supraventricualar extrasistoles due to atrioventricular nodal reentry, shown as red, demonstraining unexplained onset and stop of the episode in paroxysmal supraventricular tachycardia among normal heart rhythm shown as blue"?
- Your edit summary tells "minor ce", but it does not explain why removing this explanation is to be removed? Maxim Masiutin (talk) 11:50, 15 November 2023 (UTC)
- All I removed was "Lead II" which is not understandable to a general reader.--Iztwoz (talk) 19:20, 15 November 2023 (UTC)
- There were already "Lead" mentioned in another image in this reference. I hope that with a hyperlink it will be easier to understand when "Lead" has a wikilink, OK? Maxim Masiutin (talk) 19:23, 15 November 2023 (UTC)
- All I removed was "Lead II" which is not understandable to a general reader.--Iztwoz (talk) 19:20, 15 November 2023 (UTC)
- I put wikilink to Lead II so now it would be easier to understand the term by clicking the wikilink Maxim Masiutin (talk) 12:30, 15 November 2023 (UTC)
- Per your request, I am more than happy to take a look at this section - cheers.
- Dr. BeingObjective (talk) 23:03, 17 November 2023 (UTC)
- Have made a few edits but I am no expert. Best Iztwoz (talk) 11:28, 15 November 2023 (UTC)
A review of the Anatomy Section as requested.
editI took a look at the section, and it is fundamentally accurate.
It is vague in the 're-entrant language' - 'two path' dialog though.
I would offer the following commentary - the subject matter language is likely all about your intended target audience really is.
I offer these comments in good faith, and as stated - the anatomy section is essentially accurate - though it is written to an audience of a certain type of Cardiologist and not to who I would have considered the standard WP reader - I'd think more likely to be a medical student or a patient. Perhaps some cardiac nursing staff/NPs/PAs.
I doubt a typical GP/PCP would be in tune with this subject matter - they may have exposure to it when they were younger -
SImplistically, the assumption is that any general reader knows the following -
- the basic electrophysiology of the heart - this is in many articles - but knowing this, makes a lot of the described material a little more consumable - the writer makes the assumption that any general reader already knows the electrophysiology of a single heartbeat (a heartbeat originates from an area of tissue in the heart called the sinoatrial node (SA node). The SA node is in the right upper chamber of the heart or the right atrium. Usually the SA node creates electrical impulses around 60 to 100 times per minute. The two upper heart chambers are stimulated first followed by the two lower large ventricle chambers. 2. Electrical impulses originating from the SA node use tissue based electrical circuits to stimulate areas of the heart in a well-regulated manner and are made up of specialized cardiac tissue – these are anatomically composed of a number of conduction circuits starting in the right atrium: a. The anterior internodal tract b. The middle internodal tract c. The posterior internodal tract d. A fourth electrical tissue bundle projects from the right atrium to the left atrium and is called Bachman’s bundle. 4. From the AV node two large electrical circuits propagate into the lower chambers and are called the left and right bundles and these provide pathways for electrical stimulation of the two large lower ventricles of the heart. Just below the AV node is an area of tissue called the 'bundle of His. 5. These tracts of tissue can be thought of as electrical wiring within the heart, but this is a simplistic analogy as the circuits are often not cleanly defined as would a piece of electrical wiring in a machine.)
From some very old personal notes.
I am sure a single image would be enough and there are several floating around - but the AV node/tissue zones role in creating/controlling/regulating heart rhythms is central to the discussion IMHO.
The key part of this article that is a serious challenge to explain - the language/link that talks about a defect in the 'electrophysiology tissues' that surround/are broadly part of the AV node - the two pathways and the potential pathologies related to these. The linked article uses the same set of assumptions - so I am not clear it offered more clarity than this article.
I think my added thoughts are:
A. add content that really digs into the electrophysiological function of the AV node - is worth an effort to set the initial stage.
B. this allows for the expansion on dialog on the slow/fast circuits in a clear manner - there is a nice two-circuit image in the linked article - but again it assumes a base of knowledge that just might not be there.
It is a fairly tough subject to describe - and I am not sure I have been very helpful - I think the target audiences and the expansion of the electrophysiological role of the AV node would seriously help.
It does read like an advanced cardiology text - if this is your audience, it is likely fine.
Really sorry I could not be of more help.
BeingObjective (talk) 17:48, 18 November 2023 (UTC)
- Great! Thank you very much for your excellent review!
- I was particularly worry about the image and the description of the image (image caption) because I created the image and the description myself, not being a cargiologist, even no medical education.
- I understand that the description of reentry is not adequate for general audience. Maybe an image would have been better.
- I tried to explain to general audience the following: "There are two pathways: fast and slow; probably only fast pathway is activated in normal conditions; but if the slow pathway is also activated, than we get an onset of supraventricular tachycardia which can be as small as one, two or three heartbeats, or may last for hours".
- Thank you very much for your contributions, you are very helpful in affirming that technically this section is correct.
- We will be able to simplify this section in the future, by adding explanations and diagrams.
- But as for now, it is very good that it is correct and does not need to be deleted or corrected.
- Thank you very much again!
- I don't have any idea at the moment on how to improve the article or this section. Should an idea come, I will edit it. You can also edit it any time you find reasonable. Maxim Masiutin (talk) 22:25, 18 November 2023 (UTC)
- Maybe we can even improve the readability of the section with minimal edit, with such things as bullets, proper paragraph separations, or subsections. I initially wanted to make bullets for fast and slow pathways, but the information about them intertwines, so didn't find how to do that. Maxim Masiutin (talk) 22:27, 18 November 2023 (UTC)