Mahmud Barmada, known as Michael Barmada, (1969–2016) was an American geneticist. He was a Distinguished Professor at the University of Pittsburgh School of Medicine.[1][2]

Michael Barmada

Career edit

Barmada worked at the Institute for Personalized Medicine as associate director and co-director of the Center for Simulation and Modeling. He held a secondary appointment in the Department of Biomedical Informatics.

In 1991, he graduated from Carnegie Mellon University with a bachelor's degree in chemistry and biological sciences with a minor in piano composition; in 1993, he earned a master's in molecular genetics at Johns Hopkins; and in 1999, he earned a PhD in human genetics with a concentration in statistical and computational genetics at Pitt.

Professor Dan Weeks said, “He did more than his fair share, freely and of his own initiative, to the benefit of the department and the whole school.”[1][3]

Weeks said Barmada was among the university's first researchers to tackle next-generation sequencing. “He was asked so much for help that he created and taught a workshop on it.”[1]

According to Google Scholar, Barmada has an h-index of 56.[4] According to the Scopus he has an h-index of 48.[5]

Selected papers edit

  • A Genome-Wide Association Study Identifies IL23R as an Inflammatory Bowel Disease Gene [6]
  • Genome-wide association defines more than 30 distinct susceptibility loci for Crohn's disease[7]
  • Genome-wide association study identifies new susceptibility loci for Crohn disease and implicates autophagy in disease pathogenesis[8]

References edit

  1. ^ a b c Webteam, University of Pittsburgh University Marketing Communications. "University Times » M. Michael Barmada". Retrieved 2023-05-23.
  2. ^ Prohaska, Thomas R.; Anderson, Lynda A.; Binstock, Robert H. (2012-03-15). Public Health for an Aging Society. JHU Press. ISBN 978-1-4214-0535-3.
  3. ^ Liu, Yu (2014-02-24). Bioinformatics: The Impact of Accurate Quantification on Proteomic and Genetic Analysis and Research. CRC Press. ISBN 978-1-77188-019-0.
  4. ^ "Michael Barmada". scholar.google.com. Retrieved 2023-05-23.
  5. ^ "Scopus preview – Barmada, M. Michael – Author details – Scopus". www.scopus.com. Retrieved 2023-05-23.
  6. ^ Duerr, Richard H.; Taylor, Kent D.; Brant, Steven R.; Rioux, John D.; Silverberg, Mark S.; Daly, Mark J.; Steinhart, A. Hillary; Abraham, Clara; Regueiro, Miguel; Griffiths, Anne; Dassopoulos, Themistocles; Bitton, Alain; Yang, Huiying; Targan, Stephan; Datta, Lisa Wu (2006-12-01). "A genome-wide association study identifies IL23R as an inflammatory bowel disease gene". Science. 314 (5804): 1461–1463. doi:10.1126/science.1135245. ISSN 1095-9203. PMC 4410764. PMID 17068223.
  7. ^ Barrett, Jeffrey C.; Hansoul, Sarah; Nicolae, Dan L.; Cho, Judy H.; Duerr, Richard H.; Rioux, John D.; Brant, Steven R.; Silverberg, Mark S.; Taylor, Kent D.; Barmada, M. Michael; Bitton, Alain; Dassopoulos, Themistocles; Datta, Lisa Wu; Green, Todd; Griffiths, Anne M. (August 2008). "Genome-wide association defines more than 30 distinct susceptibility loci for Crohn's disease". Nature Genetics. 40 (8): 955–962. doi:10.1038/ng.175. ISSN 1546-1718. PMC 2574810. PMID 18587394.
  8. ^ Rioux, John D.; Xavier, Ramnik J.; Taylor, Kent D.; Silverberg, Mark S.; Goyette, Philippe; Huett, Alan; Green, Todd; Kuballa, Petric; Barmada, M. Michael; Datta, Lisa Wu; Shugart, Yin Yao; Griffiths, Anne M.; Targan, Stephan R.; Ippoliti, Andrew F.; Bernard, Edmond-Jean (May 2007). "Genome-wide association study identifies new susceptibility loci for Crohn disease and implicates autophagy in disease pathogenesis". Nature Genetics. 39 (5): 596–604. doi:10.1038/ng2032. ISSN 1061-4036. PMC 2757939. PMID 17435756.

External links edit